Peer-reviewed veterinary case report
Dog with uneven neurological signs and low blood sugar from adrenal
By Morris, Cameron A D & Donaldson, Rebekah E·Published in Journal of veterinary emergency and critical care (San Antonio, Tex. : 2001)·2025·Critical Care Department, Australia·View original on PubMed →
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Original publication title: Asymmetric Multifocal Neurological Signs in a Dog With Eunatremic, Eukalemic Hypoadrenocorticism With Severe Hypoglycemia.
- Species:
- dog
Plain-English summary
A 7-year-old neutered male miniature Poodle was brought to the vet after collapsing, vomiting, and showing unusual neurological signs like a head tilt. The dog was in shock and had low blood sugar, but tests showed no issues with the brain structure. After treatment with glucose and a steroid called dexamethasone, the dog's condition improved slowly over a couple of days. By the time he was rechecked a week later, he had mostly recovered and was back to normal.
People also search for: dog collapse and vomiting · miniature Poodle neurological signs · hypoadrenocorticism treatment in dogs
Abstract
OBJECTIVE: To describe the clinical presentation of a dog with eunatremic, eukalemic hypoadrenocorticism (EEH) with multifocal, asymmetrical neurological signs. CASE SUMMARY: A 7-year-old neutered male miniature Poodle presented for collapse, vomiting, and acute onset of neurological signs. The dog was obtunded and nonambulatory, with a right-sided head tilt and turn with rotary nystagmus. No seizure activity was observed by the owner. The patient was in hypovolemic shock. Hypoglycemia, mild azotemia, and metabolic acidosis characterized by hyperlactatemia were identified. Magnetic resonance imaging of the brain and CSF analysis performed within 24 h of presentation were unremarkable. ACTH stimulation testing confirmed hypoadrenocorticism. Neurological signs failed to significantly improve despite systemic stabilization, glucose supplementation, and dexamethasone administration until approximately 48 h following admission. A mild proprioceptive ataxia was present at discharge approximately 84 h following admission and had resolved by the time of initial recheck 7 days following discharge. NEW INFORMATION PROVIDED: EEH can cause neurological signs with forebrain localization that are typically symmetrical due to the global nature of the injury. To the authors' knowledge, this is the first report of a dog with EEH presenting with severe, asymmetric, and multifocal neurological signs.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/40884765/