Peer-reviewed veterinary case report
Mitral valve and heart changes in cats with hypertrophic
By Schober, Karsten & Todd, Amanda·Published in Journal of veterinary cardiology : the official journal of the European Society of Veterinary Cardiology·2010·Department of Veterinary Clinical Sciences, United States·View original on PubMed →
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Original publication title: Echocardiographic assessment of left ventricular geometry and the mitral valve apparatus in cats with hypertrophic cardiomyopathy.
- Species:
- cat
Plain-English summary
A group of cats with hypertrophic cardiomyopathy (HCM), a heart condition that causes thickening of the heart muscle, was examined to see how their mitral valve might be affecting their heart function. The study found that cats with HCM and obstruction had more significant changes in their heart structure compared to those without obstruction. Specifically, they had thicker heart muscles and longer mitral valve leaflets, which were linked to more severe heart issues. These findings suggest that problems with the mitral valve are common in cats with HCM and may contribute to heart problems.
People also search for: cat heart disease symptoms · hypertrophic cardiomyopathy in cats · mitral valve problems in cats
Abstract
OBJECTIVES: This retrospective study addressed the general hypothesis that abnormalities of the mitral valve apparatus are common in cats with idiopathic hypertrophic cardiomyopathy (HCM) and contribute to dynamic obstruction of the left ventricular outflow tract (LVOT). ANIMALS, MATERIALS AND METHODS: 106 cats (28 controls and 78 with HCM) had transthoracic two-dimensional and Doppler echocardiography performed with quantification of 33 variables. Three groups of cats (control [Group-1], HCM without obstruction [Group-2], and HCM with obstruction [Group-3]) were identified and compared by analysis of variance, chi(2) analysis, and correlation analysis. RESULTS: Cats in Group-3 had more LV and papillary muscle hypertrophy, increased length of the anterior mitral valve leaflet, and a higher prevalence of false tendons in the LVOT compared to cats in Group-2 (P < or = 0.05). The length of the anterior mitral valve leaflet was correlated to the severity of dynamic obstruction (P < or = 0.05) and the magnitude of LV hypertrophy (P < or = 0.001). Systolic anterior motion of chordae tendineae (CAM) was observed in 16% of control cats and >50% of cats with HCM (P < or = 0.05). CONCLUSIONS: Abnormalities of the mitral valve are common in cats with HCM suggesting a possible role in the pathogenesis of dynamic outflow tract obstruction.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/20185379/