Peer-reviewed veterinary case report
Hereditary fragmented coronoid process causing elbow lameness in dogs
By Temwichitr, Jedee et al.·Published in Veterinary journal (London, England : 1997)·2010·Department of Clinical Sciences of Companion Animals, Netherlands·View original on PubMed →
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Original publication title: Fragmented coronoid process in the dog: a heritable disease.
- Species:
- dog
Plain-English summary
A Rottweiler or Labrador Retriever with front leg lameness may be suffering from a condition called fragmented coronoid process (FCP), which is a common issue in medium-to-large breed dogs. Unfortunately, dogs with FCP often develop arthritis in the elbow joint, and treatments, whether conservative or surgical, may not fully resolve the problem. This condition is believed to be hereditary, but the specific genes involved have not yet been identified. If your dog shows signs of lameness, it's important to consult your veterinarian for a proper diagnosis and treatment options.
People also search for: dog front leg lameness · Rottweiler elbow dysplasia treatment · Labrador Retriever arthritis symptoms
Abstract
Fragmented coronoid process (FCP) is one of the main diseases associated with elbow dysplasia. FCP is often diagnosed in medium-to-large breed dogs with front leg lameness, for instance in Rottweilers, Labrador Retrievers and Bernese Mountain dogs. Dogs with FCP develop osteoarthrosis of the elbow joint despite conservative or surgical treatment. Although FCP is considered a hereditary condition, the gene or genes causing FCP have yet to be identified. This article provides an overview of different aspects of FCP, including elbow joint development, hypotheses about disease pathogenesis, the genetic background of FCP, and genetic methodology to identify gene or genes responsible for FCP.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/19640749/