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Peer-reviewed veterinary case report

Glutamate release and uptake processes are altered in a new mouse model of amyotrophic lateral sclerosis.

Journal:
Doklady. Biochemistry and biophysics
Year:
2016
Authors:
Grigoriev, V V et al.
Affiliation:
Institute of Physiologically Active Compounds
Species:
rodent

Abstract

In this paper, we showed that in the cortex of mice expressing an abberant form of FUS protein that model amyotrophic lateral sclerosis (ALS), the processes of KCl-induced and basal [(3)H]glutamate release and uptake are altered at the presymptomatic stage as compared to the non-transgenic littermates. The change in these three parameters in transgenic animals causes excitotoxicity, which, in turn, may lead to massive loss of motor neurons and the onset of ALS symptoms.

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Original publication: https://pubmed.ncbi.nlm.nih.gov/27417710/