Peer-reviewed veterinary case report
Dog develops brain damage and neurological signs days after smoke
By Weiss, A Th A et al.·Published in Veterinary pathology·2011·Department of Veterinary Pathology, Germany·View original on PubMed →
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Original publication title: Leukoencephalomalacia and laminar neuronal necrosis following smoke inhalation in a dog.
- Species:
- dog
Plain-English summary
A dog developed serious breathing and neurological problems six days after being rescued from a fire. The symptoms included progressive neurological issues, which were linked to damage in the brain caused by smoke inhalation. Unfortunately, the dog did not survive, and a post-mortem examination revealed significant brain damage. This case highlights the potential for delayed neurological symptoms in pets exposed to smoke.
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Abstract
Acute respiratory and neurologic disease after smoke inhalation are well documented, but human patients may also develop delayed-onset neurologic symptoms associated with leukoencephalomalacia after exposure to smoke or carbon monoxide. In this case, a dog developed progressive neurologic signs 6 days after rescue from an apartment fire. At necropsy 9 days after smoke inhalation, leukoencephalomalacia of the central cerebral white matter was accompanied by laminar necrosis of cerebrocortical neurons. This is the first report of delayed posthypoxic leukoencephalopathy in a nonhuman animal.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/20940447/