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Peer-reviewed veterinary case report

PARK2/Parkin becomes critical when DNM1L/Drp1 is absent.

Journal:
Autophagy
Year:
2015
Authors:
Roy, Madhuparna et al.
Affiliation:
a Department of Cell Biology · United States

Abstract

Maintaining mitochondrial dynamics and proper execution of mitophagy is crucial for sustaining cellular health. Defects in these processes have been linked to cardiovascular diseases and neurodegeneration. In a recent publication, we reported that the mitochondrial division dynamin protein DNM1L/Drp1 and the E3 ubiquitin ligase PARK2/Parkin work in a synergistic manner to maintain mitochondrial function and structural integrity in the mouse heart and brain.

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Original publication: https://pubmed.ncbi.nlm.nih.gov/25715097/