Peer-reviewed veterinary case report
Mitral valve motion linked to right heart pressure in 9 dogs
By Paige, Christopher F et al.·Published in Journal of veterinary cardiology : the official journal of the European Society of Veterinary Cardiology·2007·Department of Small Animal Clinical Sciences, United States·View original on PubMed →
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Original publication title: Systolic anterior motion of the mitral valve associated with right ventricular systolic hypertension in 9 dogs.
- Species:
- dog
Plain-English summary
Nine dogs were diagnosed with a heart condition called systolic anterior motion of the mitral valve (SAM), which was linked to high blood pressure in the right side of the heart. Some of these dogs had other heart issues, like pulmonic stenosis or pulmonary hypertension. While the heart's structure showed some abnormalities, the overall impact on their health seemed minor. Treatment details weren't specified, but monitoring and managing the underlying heart conditions would be important for these pets.
People also search for: dog heart problems · mitral valve issues in dogs · pulmonary hypertension treatment in dogs
Abstract
OBJECTIVES: To describe the zoographic and echocardiographic characteristics of canine patients in which systolic anterior motion of the mitral valve (SAM) was identified in association with right ventricular systolic hypertension (RVSH). ANIMALS, MATERIALS AND METHODS: Medical records and digitally recorded echocardiographic examinations were reviewed for RVSH and two-dimensional (2DE) or M-mode echocardiographic evidence of SAM. RESULTS: SAM was identified in association with RSVH in 9 patients; 5 had pulmonic stenosis, 2 had tetralogy of Fallot and 2, pulmonary hypertension. Relative to body weight, the end-diastolic and end-systolic left ventricular dimensions were subnormal in all patients. Hyperdynamic left ventricular systolic performance was identified in 8 of 9 patients. In 5 of the 9 patients, SAM was mild or moderate in degree. Left ventricular outflow tract (LVOT) obstruction and mitral valve regurgitation were documented by Doppler studies in only 3 of the 4 patients with marked SAM. However, late systolic acceleration within the LVOT was recorded in 2 additional patients for whom peak velocities were normal. CONCLUSIONS: In the cases described here, the presence of SAM is likely explained by alterations in left ventricular geometry and function associated with diminished pulmonary venous return together with sympathetic activation resulting from subnormal stroke volume. Although the hemodynamic consequences were apparently minor, the association of SAM with right-sided heart disease might be of interest to those engaged in the practice of veterinary echocardiography.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/17689464/