Peer-reviewed veterinary case report
Dog developed kidney acid and diabetes insipidus after anesthesia
By Ku, Dayoung et al.·Published in Veterinary medicine and science·2023·College of Veterinary Medicine, South Korea·View original on PubMed →
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Original publication title: Transient distal renal tubular acidosis with nephrogenic diabetes insipidus after general anaesthesia in a dog.
- Species:
- dog
Plain-English summary
A 3-year-old female spayed Pomeranian was brought to the vet after experiencing persistent vomiting, not eating, excessive urination, and thirst for a week following surgery. The dog was lethargic and showed signs of dehydration. Tests revealed a kidney issue called distal renal tubular acidosis (RTA) and suspected nephrogenic diabetes insipidus (DI), which caused her to urinate excessively. The vet treated her with potassium citrate to help with the acidosis and desmopressin acetate along with hydrochlorothiazide for the diabetes insipidus. After 24 days of treatment, her symptoms improved, and she was no longer experiencing excessive urination.
People also search for: dog vomiting after surgery · Pomeranian kidney problems · diabetes insipidus treatment for dogs
Abstract
A 3-year-old, 3.5 kg, female spayed Pomeranian was referred due to persistent vomiting, anorexia, polyuria and polydipsia, 7 days after receiving general anaesthetic for a medial patellar luxation correction. Physical examination revealed lethargy, tachypnoea and 7% dehydration. Complete blood count and serum chemistry results were unremarkable, and venous blood gas analysis revealed hypokalaemia and hyperchloraemic metabolic acidosis with a normal anion gap. Urinalysis revealed a urine specific gravity (USG) of 1.005, pH of 7.0 and proteinuria, and the bacterial culture was negative. Based on these results, the dog was diagnosed with distal renal tubular acidosis, and potassium citrate was prescribed to correct metabolic acidosis. In addition, concurrent diabetes insipidus (DI) was suspected because the dog showed persistent polyuria, polydipsia and a USG below 1.006 despite dehydration. After 3 days of initial treatment, acidosis was corrected, and vomiting resolved. Desmopressin acetate and hydrochlorothiazide were also prescribed for DI, but the USG was not normalized. Based on the insignificant therapeutic response, nephrogenic DI was highly suspected. DI was resolved after 24 days. This case report describes the concomitant presence of RTA and DI in a dog after general anaesthesia.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/37224266/