Peer-reviewed veterinary case report
Primary hyperoxaluria causing weakness and kidney crystals in a young
By De Lorenzi, Davide et al.·Published in Journal of feline medicine and surgery·2005·Clinica Veterinaria S. Marco, Italy·View original on PubMed →
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Original publication title: Primary hyperoxaluria (L-glyceric aciduria) in a cat.
- Species:
- cat
Plain-English summary
A 7-month-old male European cat was brought to the vet because he was weak and not eating. He was dehydrated and had severe muscle wasting, with reduced reflexes. Tests showed high levels of oxalate in his urine and increased L-glyceric acid in his blood, indicating a serious metabolic issue. Unfortunately, further examination revealed significant damage to his kidneys and spinal motor neurons, leading to a poor prognosis. Sadly, the cat did not survive due to the severity of his condition.
People also search for: cat weakness and not eating · cat kidney disease symptoms · cat muscle atrophy causes
Abstract
A 7-month-old, male European cat was examined because of weakness and inappetence. The cat was dehydrated, polypnoeic and severely weak. Severe, generalised muscle atrophy was present. Spinal reflexes were all decreased to absent. Blood analysis and urinalysis showed several abnormalities, including intermittent hyperoxaluria. The L-gliceric acid concentration was remarkably increased. Electrodiagnostic tests of the peripheral nervous system were abnormal. At necropsy, generalised muscle atrophy was observed. Microscopically, both kidneys showed intraluminal birefringent oxalate crystals. Motor neuron degeneration and accumulation of neurofilaments were observed in the axons of the spinal motor neurons.
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Search related cases →Original publication on PubMed: https://pubmed.ncbi.nlm.nih.gov/15914058/